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Within the healthy population, there is substantial, heritable, and interindividual variability in the platelet response.

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Despite a rich literature on DNA repair processes, reconstruction of the molecular events generating each of these mutations is not yet possible. This work has led to the discovery of previously unknown, evolutionarily conserved subunits of the anaphase-promoting complex and the gamma-tubulin ring complex--large complexes that are essential for spindle assembly and chromosome segregation. Two transit compartment chains accounted for the delay between exposure and response.

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